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The bound copper is biologically active GHK-Cus signaling and regenerative effects are dependent on the copper moiety, not just the peptide

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These are consistent with the mechanism of action slowing gastric emptying affects digestion, and central appetite suppression can cause nausea, particularly during the early stages of treatment and dose escalation

Current models of APC action emphasize its role as a component of DCs to promote -catenin degradation

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The cell death induced by the B2M-ADC does not seem to be led by apoptosis but rather by necrosis 49