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All data are presented as the mean standard error of the mean (SEM)

48 Corts-RamrezD.-A.Gainza-CirauquiM.-L.Echebarria-GoikouriaM.-A

The redox imbalance is more likely induced by the net effect of low antioxidative defense systems and thus constantly produces endogenous RONS, such as angiotensin II, lipoxygenase, nicotinamide adenine dinucleotide phosphate (NADPH) oxidase, and myeloperoxidase [19]

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Given the apparent safety of reduced GSH supplementation, safety is not an apparent barrier to continued clinical trials of GSH supplementation in relevant disease states

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