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10.3322/caac.21834 11 BuchananR

16,17,18,56 Meanwhile, the improved oxygen availability alleviates the hypoxic suppression of BMAL1, thereby enabling its transcriptional activity and promoting a pro-regenerative outcome in bone regeneration

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Matrix metalloproteinase-9 activation in hyperhomocysteinemic rats is triggered by the opening of the mitochondrial permeability transition pore and antagonism of the N-methyl-D-aspartate receptor-1 (NMDA-R1), resulting in myocyte dysfunction due to elevated calcium overload and oxidative stress [40]

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Clinical studies have indicated that liraglutide can reduce metabolic dysfunction, insulin resistance and lipotoxicity in patients with NASH without improving fibrosis [102, 103]