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In humans, GWAS is the main-stream analysis platform for polygenic traits like idiosyncratic DILI, for which the effect size of a polymorphism, e.g., single nucleotide polymorphism (SNP), is often small while a large number of loci could be associated with the trait [33]

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Clinician insight: oxidative stress markers (F2-isoprostanes, 8-OHdG), mitochondrial assays, and low-grade immune markers are rarely measured despite being early indicators of damage