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Most studies focus on short-term use 2

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5 Reactive oxygen species as oxidizing agents: impact on cysteine proteinases and cellular homeostasis It has long been established that disturbances in reactive oxygen species (ROS) regulation generate highly reactive cellular environments, profoundly influencing intracellular signaling pathways and contributing to oxidative stress

Particularly noteworthy, mechanical overload can activate the mechanosensitive ion channel Piezo1, mediating calcium influx, which subsequently triggers the downstream GPX4-regulated ferroptosis pathway (Yao et al., 2021

Non-mitochondrial ROS sources in diabetes Regardless of whether excessive ROS formation is the primary initiator or accompanying factor in the pathogenesis of diabetes mellitus, oxidative stress plays a key role in the organ complications such as cardiovascular problems, neurological problems (stroke), diabetic retinopathy, nephropathy, and neuropathy

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