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Caspase-3-mediated death of melanocytes can also be triggered by ROS-induced secretion of adenosine triphosphate from keratinocytes, which can also induce nearby keratinocytes to produce CXCL9 to chemically attract CD8+ T cells [24]

This combination also manifest synergistic efficacy in attenuating the HOMA-IR, implying complementary mechanisms in addressing key metabolic and endocrine disturbances in PCOS (182)

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The greatest efficacy in their TMZ-resistant orthotopic GBM model was observed when comparing OS between TMZ and TMZ plus their HDAC inhibitor (MPT0B291) (130)

Montesinos MC, Desai D et al (2003): Adenosine A2A or A3 receptors are required for inhibition of inflammation by methotrexate and its analog MX-68

[44] This gives a plausible mechanism for cells to couple oxidative stress signals with cellular mainstream signaling such as phosphorylation