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This review focuses on the relationship between AMS and hemodynamic and hemorheological stress, changes in cytokines and inflammatory cytokines, regulation of related signaling pathways, blood-brain barrier damage, oxidative stress injury, autophagy, and summarises the establishment of AMS animal model methods

Ninety-seven percent had ascites, 64% had hepatic encephalopathy, 9% had portal hypertensive bleeding, and 58% met the NACSELD definition of ACLF

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Gan WQ, Man SF, Senthilselvan A, Sin DD

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