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Furthermore, the administration of GSH and Fer-1 treatment to S8/ Gss / mice ameliorated testicular defects by inhibiting ferroptosis

The coupled systems architecture reveals that oxidative stress and C1 metabolism interface through the ascorbate-glutathione pathway, which interfaces with the methionine biosynthesis and formaldehyde detoxification pathways of C1 metabolism

Interestingly, an increase in reproductive fitness (Fig

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