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Promisingly, in-vivo oximetry (as a combination of different MRI modalities, such as arterial spin labelling and BOLD imaging, susceptibility-weighted imaging, or 17 O-MRI) shows potential for discerning in situ mitochondrial impairment (Borghammer et al., 2008)

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Because pronounced responsiveness of roGFPs is limited to a range of about 35 mV around their midpoint redox potentials, it can be concluded that the actual E GSH in the ER lumen is at least 35 mV less negative than the midpoint potential of roGFP2 (Mller-Schssele et al., 2021)

They accumulate in various tissues, e.g., in the thymus (Muller, 1977), uterus (Gouon-Evans, 2001), lungs (Mesnil et al, 2016), adipose tissue (Wu et al, 2011), mammary gland (Gouon-Evans et al, 2000) and gastrointestinal tract (Chu et al, 2014), whereas the peripheral blood contains only few eosinophils under homeostatic conditions