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Description

For patients receiving B12 replacement therapy for pernicious anaemia or deficiency , monitoring is typically arranged by the prescribing clinician

F1534C mutations were detected in permethrin-resistant Ae

The abnormal aggregation of -syn can disrupt mitochondrial homeostasis through several mechanisms: (1) Inhibition of mitochondrial membrane fusion: In vitro experiments have demonstrated that the direct interaction of -syn with the mitochondrial membrane can lead to mitochondrial fragmentation and activation of the apoptotic pathway (Panicker et al., 2021)