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Inhibition of monoamine oxidase B prevents reactive astrogliosis and scar formation in stab wound injury model

10.1016/0169-328X(93)90072-W Mol

Summary Keywords rheumatoid arthritis, reactive oxygen species, mitochondrial damage, targeted drugs, oxidative stress Citation Jing W, Liu C, Su C, Liu L, Chen P, Li X, Zhang X, Yuan B, Wang H and Du X (2023) Role of reactive oxygen species and mitochondrial damage in rheumatoid arthritis and targeted drugs

This reaction is reversible and can adapt to higher levels of homocysteine, converting it back to SAH

Recent investigations have unearthed a novel player in the ferroptosis domain, MBOAT2, functioning autonomously of GPX4 and FSP1, overseeing ferroptosis surveillance through phospholipid remodeling and proficiently impeding ferroptotic processes

Hormone molecular biology and clinical investigation