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Silencing UCP2 significantly inhibits cancer cell proliferation, highlighting UCP2 as a potential therapeutic target [137]

Hypothetically, the increase in saliva melatonin may be associated with the activation of the serotonergic system during slow wave sleep suppression [209]

Perhaps the most striking property is GHK-Cus effect on gene expression

Both the absence and overexpression of MECP2 lead to neurofunctional deficits akin to those observed in RTT patients, emphasizing the need for careful control of MECP2 expression (116, 117)