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Over the past few decades, copper-induced cell death, named cuproptosis, has become increasingly recognized as an important process mediating the pathogenesis and progression of cardiovascular disease (CVD), including atherosclerosis, stroke, ischemia-reperfusion injury, and heart failure

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One fragment to highlight is x2267, which is bound to site S12, 6.36 away from Asn363 residue in the insertion (359363) (Fig

Wadley AJ, Keane G, Cullen T et al (2019) Characterization of extracellular redox enzyme concentrations in response to exercise in humans