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However, the interactions and synergies between these mechanisms and their changing patterns in NAFLD development require further clarification

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Early-stage research suggests that luteolin may elevate the levels of brain-derived neurotrophic factor (BDNF) and amplify the downstream tropomyosin receptor kinase B signaling, which is instrumental in nurturing dendritic spine upkeep, promoting synaptic protein expression, and facilitating processes tied to long-term potentiation ( 3.4 Anti-apoptotic and anti-ferroptotic effects Luteolin exerts neuroprotective effects against programmed cell death by modulating both intrinsic and extrinsic pathways