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(4) It dephosphorylates IL-23, failing to bind to the IL-17 gene promoter to inhibit downstream IL-17 transcription [178]

Future research should concentrate on the clinical translation of the technology and handle safety, long-term efficacy, and ethical issues

Inflammation, ectopic lipid deposition, endoplasmic reticulum stress (ERS), and oxidative stress are involved in the onset and progression of T2D and TOD by impairing insulin sensitivity and/or cell dysfunction, reciprocal with metabolic disorders

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