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The researchers continue: In pernicious anemia [B12] absorption is impaired due to IF deficiency arising from autoimmune destruction of parietal cells

This sustained-release property can be beneficial for individuals who require less frequent injections or treatments

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For example, in the Inflammasome pathway , NLRP1 was inhibited in the MOD group and B12 abrogates this inhibition ( Figure S8 )

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