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In the case of glucocorticoids, both excess (Cushings syndrome) and insufficiency (Addisons disease) contribute to oxidative stress through a variety of mechanisms, such as decreased antioxidant defenses, increased reactive oxygen species (ROS) generation, and impaired mitochondrial function, which ultimately results in chronic inflammation

To integrate samples and mitigate batch-associated structure, we applied Harmony on the reduced-dimensional representation prior to downstream clustering and visualization 34

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YTHDF1 knockdown significantly increased the survival rate of KGN cells, but SLC7A5 overexpression effectively attenuated this protective effect (Fig