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Regardless of the origin of PKC overexpression, it is clear that this protein is emerging through the literature as an important biomarker and potential drug target for many cancer types

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It is tempting to speculate that the stimulus-specific differences in PKC phosphorylation profiles (that impact on substrate specificity) underlie PKCs diverse roles in both the pathogenesis of ischemia-reperfusion injury and as a mediator of ischemic preconditioning (Steinberg, 2012)

Some of the dynamical coefficients values were estimated from experimental data such as degradation rates, and rates of constitutive and autophosphorylations

There are also studies claiming the opposite (Reference Rossi, Abusief and Missmer61)

Realworld scenarios clinicians see Real patients and trial participants add nuance