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Collectively, these pathways converge to cause sperm mitochondrial dysfunction, increased DNA fragmentation, and reduced motility

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Wound contraction was accelerated due to increased migration of myofibroblasts, fibroblasts, and macrophages within wounds treated by curcumin (Sidhu et al., 1998)

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To prevent excessive activation, complement is strongly controlled by C1-INH (CP), FH and FHL-1 (AP), and C4BP (CP and LP), vitronectin (Vn) clusterin, and FHR-1 (TP) (Fig

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