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Establishment and characterization of MEC-1/N M and MEC-1/N WT MEC-1 models recapitulating CLL metabolic signatures To overcome CLL cell constraints, we developed a cellular model carrying unmutated BCR with or without NOTCH1 mutations

202 However, after the end of treatment with diazoxide and alkylating agents, tumors in this group grew faster than in controls

At the same time, mitochondrial dysfunction is ameliorated via promoting mitochondrial biogenesis to inhibit abnormal neuronal apoptosis (Zheng et al., 2023)