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354 Overall, therapeutic approaches involving drugs, physical treatment, or gene modifications enhancing AKT-related signaling pathways and NMDAR activities could reinforce synaptic NMDAR activities and their neuroprotective effects in ischemic stroke

and 6)ischemia resulting from transient four-vessel occlusion (reviewed in [45])

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In short, a NRTI-induced decrease in mtDNA leads to malfunctioning of mitochondrial protein complexes and changes in respiration rate, decreased ATP production, a diminished mitochondrial membrane potential, and an escalation in ROS production (Lewis et al., 2006