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Results C57 mice were randomly grouped and exposed to normoxic or hyperoxic conditions, respectively, and the ratio of reduced glutathione GSH/oxidized glutathione was decreased in hyperoxia, and high expression of GSH synthase (Gss) was found

1dg, the glycemia of THR-123-treated animals, regardless of the dose, was significantly lower than that of saline-treated mice throughout the experiment ( p 0.05)

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2C, S2C)

Additionally, another study confirmed that CU significantly inhibited the overexpression of monocyte chemoattractant protein-1 (MCP-1) and tumor necrosis factor-alpha (TNF-), reduced lipid peroxidation, and restored MDA levels, which collectively ameliorated neurological deficits, alleviated cerebral vasospasm, and significantly decreased mortality in SAH (Luo et al., 2011)

doi: 10.1016/j.canlet.2014.12.015