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Oxid Med Cell Longev (2021) 2021:6617256

More specifically, the researchers posited that the peptide may have enhanced the expression of Ki-67 and CD98hc that are less intensively synthesized during cell aging. Epithalon also seemed to inhibit the activity of Caspase-3, a crucial enzyme believed to facilitate apoptosis (programmed cell death)

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doi: 10.1016/j.ejmech.2019.01.078

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Antioxidants 10(7):1069 Sun Y, He L, Wang T, Hua W, Qin H, Wang J et al (2020) Activation of p62-Keap1-Nrf2 pathway protects 6-hydroxydopamine-induced ferroptosis in dopaminergic cells