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Int J Gen Med 17:37193731

The first evidence that eNOS uncoupling may be a pathophysiologically relevant phenomenon was provided by in vitro studies in which it was demonstrated that native low-density lipoprotein 81 and even more pronounced oxidized low-density lipoprotein 82 are able to stimulate endothelial superoxide production and that this phenomenon is inhibited by the NOS inhibitor N G -nitro-L-arginine methyl ester, pointing to a causal role for eNOS in superoxide production

Consequently, this leads to a reduction in ROS production, MDA levels, and H 2 O 2 content, thereby maintain cellular membrane integrity

Chen Y, Ding SX, Zhang H, Sun ZH, Shen XY, Sun LL, et al

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