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It disrupts the inhibitory interaction between DPP8/9 and the inflammasome sensor NLRP1 (NLR family member, pyrin domain containing 1), leading to NLRP1 inflammasome formation, and subsequently cell death

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This leads to a fundamental unresolved question: Do specific microbial changes precede and potentially contribute to AD pathology, or do they arise as a result of the disease state itself

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*Correspondence: Lidiia Mys, [email protected] This article was submitted to Mitochondrial Research, a section of the journal Frontiers in Physiology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers