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During latency only a few essential viral genes are expressed and production of virions are stalled ( + or EBV tumors are typically subjected to similar treatment regimen

Consistent with reports showing that the topoisomerase II-inhibitor Etoposide is a substrate of ABCC1 42 , the GI 50 of MOLM-13 ABCC1 -knockout clones to Etoposide was 1.4 2.4-fold lower than in control cells (Supplementary Fig

doi: 10.1097/MCC.0000000000000510 238 MotkowskiRMaciejczykMHryniewickaMKarpiskaJMikouB

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This, in turn, affects enolase 1 (ENO1)-mediated glycolysis, ultimately contributing to the development of myocardial fibrosis (58)

The formulation was co-developed by Dr