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(2019) 14:e0213060

The RIPK1/RIPK3/MLKL complex induces necroptosis through multiple mechanisms, including promoting mitochondrial autophagy, producing mitochondrial reactive oxygen species, and altering mitochondrial permeability, leading to mitochondrial dysfunction (141)

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Immunohistochemistry of brain tissues from mice treated with GHK showed evidence of decreased inflammation and increased labeling of histone deacetylase 2, suggesting that in addition to an anti-inflammatory effect, GHK may be triggering an epigenetic pathway in the amelioration of cognitive impairment in aging mice