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Carefully targeting specific metabolic branches within the arginine network may offer promising therapeutic avenues for IPF, allowing modulation of fibrosis while preserving essential immune and vascular functions

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Enhanced half-life of genetically engineered human IgG1 antibodies in a humanized FcRn mouse model: potential application in humorally mediated autoimmune disease

HEK293 cells expressing Chac1-Myc were treated with MG132, lysosomal acidification inhibitors (Bafilomycin A1 (Baf) or Concanamycin A (CMA)), or the calpain I inhibitor N -Acetyl-L-leucyl-L-leucyl-L-norleucinal (ALLN)