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Murakami S, Kondo Y, Sakurai T, Kitajima H, Nagate T

Salhan D, Pathak S, Husain M et al (2012) HIV gene expression deactivates redox-sensitive stress response program in mouse tubular cells both in vitro and in vivo

This was a 0.03% solution of H202

On the one hand, the increase in SOD3 activity promoted the accumulation of nitric oxide around blood vessels and reduced vascular leakage by inducing the transcription of vascular endothelial cadherin (VEC)

As a consequence, cell volume increased markedly (Figure 3C)