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LPS binds to Toll-like receptor 4 (TLR4) on the surface of Kupffer cells, activating both the MyD88-dependent pathway, which mediates NF-B signaling, and the MyD88-independent pathway involving interferon regulatory factor 3 (IRF3), ultimately upregulating TNF- transcription [49, 50]

[Google Scholar] Transcriptional control of drug resistance, virulence and immune system evasion in pathogenic fungi: A cross-species comparison

J Pharmacol Exp Ther (2002) 301(2):41826

Of note, I have fibromyalgia and an autoimmune disease (ankylosing spondylitis)

Mattson et al., 2018)

The ENS affects bone by secretion of neuropeptides like substance P, which may enhance bone formation (274, 275), and secretion of molecules like vasoactive intestinal peptide, which may suppress osteoclast activity and bone resorption (276)