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In addition, we observed that the number of activated microglia/macrophages at the injury site in the CD38-deficient mice was significantly lower than that in the WT mice, while CD38 was expressed in brain microglia

Audia, S

Methacrylic anhydride was used in this study to chemically modify chitosan and gelatin to develop methacrylate chitosan (ChiMA) and methacrylate gelatin (GelMA), respectively, to construct bridging defects on the visco-elastic skeleton

This response is often attributed to the profound immunosuppression exerted by MDSCs within the TME, which can limit the infiltration and activity of effector immune cells [6]

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