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Wang Q, Tian J, Li X, Liu X, Zheng T, Zhao Y, Li X, Zhong H, Liu D, Zhang W, et al

Parkin interacts with Ambra1 to induce mitophagy

Jeong Y-J, Kang M-J, Lee S-J, Kim C-H, Kim J-C, Kim T-H, et al

Sustained catabolism, rapid skeletal muscle loss, and wound-healing demands often justify higher protein targets, particularly in major burns and selected high-risk critically ill populations (2, 3, 51, 56, 67, 124)

For instance, homocarnosine acts as a protective agent against a wide range of disease conditions, including the protection of brain endothelial cells from amyloid peptide-induced toxicity 123 and anti-inflammatory action in brain ischemic injuries 124

Abstract Parkinsons disease (PD) is a neurodegenerative condition marked by a steady loss of dopaminergic neurons in the brains substantia nigra