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Furthermore, under OA-related inflammatory and metabolic stress environments, cells may promote GPX4 ubiquitination and subsequent proteasomal degradation by inducing specific E3 ubiquitin ligase activity, thereby increasing cellular susceptibility to ferroptosis (Su et al., 2025

Patent CN114246861A focuses on the preparation method of shear stress-responsive drug-loaded nanoparticles designed for the treatment of atherosclerosis (Li et al., 2022)