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Gill HS, Denson DD, Burris BA, Prausnitz MR

2020 Jan;39(1):110-122

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Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]

Lukcs et al., 2019), a proline rich region, a MT recognizing and binding domain, and finally the C-terminal region (Brandt et al., 2020)

This can develop in people who are taking insulin or other medications for diabetes, such as sulfonylureas, and people with serious blood glucose imbalance and fluctuations