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A growing body of evidence suggests that endothelial dysfunction is a central mechanism in COVID-19 and has also been identified as a key pathogenic mechanism in LC

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This regeneration enables it to maintain its antioxidant capacity longer than other antioxidants, which makes it crucial in protecting cells from oxidative damage over extended periods

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However, emerging findings have highlighted the contribution of resident immune cells and of immune molecules expressed by kidney-resident parenchymal cells to disease processes

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