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The authors reported in Hmox1 -deficient mice that lack of HO-1 expression in the damaged lung regions is positively related to an enhanced ROS and RNS production by neutrophils and macrophages, as well as with increased susceptibility to Mtb infections (224)

Specifically, GA significantly increased the expression of PGC1 target genes such as mitochondrial transcription factor A, nuclear respiratory factor-1, and nuclear respiratory factor-2, thereby upregulating fatty acid -oxidation enzymes like carnitine palmitoyltransferase-I, and promoting fatty acid oxidative metabolism in mitochondria (Doan et al., 2015)

2018 doi: 10.1177/1352458517711568

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Aceves, I

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